Pediatrics — Google Scholar. Fridovich I, Freeman B Antioxidant defences in the lung.
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Role of inflammation in the pathogenesis of bronchopulmonary dysplasia. J Clin Invest — Google Scholar. Moyer WT Vitamin E levels in term and preterm newborn infants. Rodriguez MP, Sosenko IRS, Antigua MC, Frank L Prenatal hormone treatment with thyrotropin releasing hormone and with thyrotropin releasing hormone plus dexamethasone delays antioxidant enzyme maturation but does not inhibit a protective antioxidant enzyme response to hyperoxia in newborn rat lung.
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Hypoxia induces severe right ventricular dilatation and infarction in heme oxygenase-1 null mice. Functional analysis of endothelial PAS domain Protein 1: Generation of a dominant negative mutant by deleting C-terminal potent transactivation domain. Inhaled nitric oxide does not affect adenosine 5'-diphosphate-dependent platelet activation in infants with persistent pulmonary hypertension of the newborn.
Hypoxic responses of vascular cells. Carbon monoxide and nitric oxide suppress the hypoxic induction of vascular endothelial growth factor gene via the 5' enhancer. Increased vascular endothelial growth factor production in the lungs of rats with hypoxia-induced pulmonary hypertension. Combination of inhaled nitric oxide with high frequency ventilation reduces the need for extracorporeal membrane oxygenation in infants with persistant pulmonary hypertension of the newborn: Results from a single center trial. Characterization of functional domains of endothelial PAS domain protein-1 and generation of a dominant negative mutant.
Carbon monoxide controls the proliferation of hypoxic vascular smooth muscle cells. Improved oxygenation in a randomized trial of inhaled nitric oxide for persistent pulmonary hypertension of the newborn. Endothelin-1 production during the acute chest syndrome in sickle cell disease.
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Effect of inhaled nitric oxide on endothelin-1 and cyclic guanosine 5'-monophosphate plasma concentrations in newborn infants with persistent pulmonary hypertension. Mechanisms by which oxygen regulates gene expression and cell-cell interaction in the vasculature. Kidney Int. Endogenous carbon monoxide regulates tube formation by human microvascular endothelial cells under high glucose. Inhaled nitric oxide does not inhibit platelet activation in infants with persistent pulmonary hypertension of the newborn PPHN. Improved oxygenation in a randomized trial of inhaled nitric oxide for PPHN.
Ketsuatsu - Japanese Journal of Blood Pressure. View abstract Morita T, Kourembanas S.
Endothelial cell expression of vasoconstrictors and growth factors is regulated by smooth muscle cell-derived carbon monoxide. Basic fibroblast growth factor increases nitric oxide synthase production in bovine endothelial cells. Am J Physiol. Hypoxia regulates vascular endothelial growth factor gene expression in endothelial cells. Identification of a 5' enhancer. Smooth muscle cell-derived carbon monoxide is a regulator of vascular cGMP. Structural characterization and specificity of expression of E2F a new member of the E2F family of transcription factors.
Cell Mol Biol Res. Increased epithelial cell expression of vascular endothelial growth factor VEGF in the lungs of animals with hypoxia-induced pulmonary hypertension. Hypoxia upregulates the transcription of human vascular endothelial growth factor gene via a bp enhancer. Smooth muscle cell-derived carbon monoxide is a regulator of endothelial cell gene expression and cGMP content.
Current Pharmacologic Approaches for Prevention and Treatment of Bronchopulmonary Dysplasia
Persistent pulmonary hypertension of the newborn: Role of nitric oxide. Care Medicine. Hypoxia increases cGMP levels in rat vascular smooth muscle cells via inducation of heme oxygenase Hypoxia inhibits expression of eNOS via transcriptional and posttranscriptional mechanisms. View abstract Kourembanas S, Bernfield M. Hypoxia and endothelial-smooth muscle cell interactions in the lung.
Transforming growth factor-beta 1, but not dexamethasone, down-regulates nitric-oxide synthase mRNA after its induction by interleukin-1 beta in rat smooth muscle cells. Nitric oxide regulates the expression of vasoconstrictors and growth factors by vascular endothelium under both normoxia and hypoxia. Hypoxia inhibits nitric oxide synthase NOS gene expression and nitric oxide NO production in endothelial cells. Tumor necrosis factor increases transcription of the heparin-binding epidermal growth factor-like growth factor gene in vascular endothelial cells.
Hypoxic responses of the neonatal endothelium. Feedback loops regulated the production of vasoactive agents by human endothelial cells. Hypoxia induces endothelin gene expression and secretion in cultured human endothelium. Molecular mechanisms contrbuting to hypoxic vasoconstriction. Oxygen tension regulates the expression of the platelet-derived growth factor-B chain gene in human endothelial cells. Dev Biol. Molecular mechanisms contributing to vaso-occlusion in sickle cell disease.
Platelet-derived growth factor production by human umbilical vein endothelial cells is regulated by basic fibroblast growth factor. Immortalization of human endothelial cells by murine sarcoma viruses, without morphologic transformation. J Cell Physiol.
Section of Neonatology, Perinatal and Pulmonary Biology
Endothelial cells synthesize basic fibroblast growth factor and transforming growth factor beta. Growth Factors. Regulation of platelet-derived growth factor PDGF gene expression by basic fibroblast growth factor bFGF in human umbilical vein endothelial cells. Increases in the 35kDa surfactant-associated protein and its mRNA following in vivo dexamethasone treatment of fetal and neonatal rats. Primary translation products, biosynthesis, and tissue specificity of the major surfactant protein in rat.